Note: This study found no significant biological effects under its experimental conditions. We include all studies for scientific completeness.
Exposure to 1950-MHz TD-SCDMA Electromagnetic Fields Affects the Apoptosis of Astrocytes via Caspase-3-Dependent Pathway.
Liu YX, Tai JL, Li GQ, Zhang ZW, Xue JH, Liu HS, Zhu H, Cheng JD, Liu YL, Li AM, Zhang Y. · 2012
View Original AbstractCell phone radiation at 1950 MHz damaged brain support cells and triggered cell death after 48 hours of exposure.
Plain English Summary
Chinese researchers exposed brain cells (astrocytes) to cell phone radiation at 1950 MHz for up to 48 hours and found that prolonged exposure damaged the cells' power centers (mitochondria) and triggered programmed cell death. While the radiation didn't promote tumor formation, it caused significant cellular damage through a specific biological pathway involving proteins that control cell death. This suggests that continuous exposure to cell phone frequencies may harm healthy brain cells even when it doesn't directly cause cancer.
Study Details
This study investigated whether EMF radiation would alter the biology of glial cells and act as a tumor-promoting agent.
We exposed rat astrocytes and C6 glioma cells to 1950-MHz TD-SCDMA for 12, 24 and 48 h respectively,...
A 48 h of exposure damaged the mitochondria and induced significant apoptosis of astrocytes. Moreove...
herefore, our results implied that exposure to the EMF of 1950-MHz TD-SCDMA may not promote the tumor formation, but continuous exposure damaged the mitochondria of astrocytes and induce apoptosis through a caspase-3-dependent pathway with the involvement of bax and bcl-2.
Show BibTeX
@article{yx_2012_exposure_to_1950mhz_tdscdma_3204,
author = {Liu YX and Tai JL and Li GQ and Zhang ZW and Xue JH and Liu HS and Zhu H and Cheng JD and Liu YL and Li AM and Zhang Y.},
title = {Exposure to 1950-MHz TD-SCDMA Electromagnetic Fields Affects the Apoptosis of Astrocytes via Caspase-3-Dependent Pathway.},
year = {2012},
url = {https://pubmed.ncbi.nlm.nih.gov/22870319/},
}