A potential multiple resonance mechanism by which weak magnetic fields affect molecules and medical problems: the example of melatonin and experimental "multiple sclerosis"
Authors not listed · 2006
The paper proposes a multiple resonance mechanism by which weak nanoTesla magnetic fields could ameliorate melatonin-related diseases.
Plain English Summary
The hypothesis predicts optimal ameliorating effects on experimental allergic encephalomyelitis at derived intensities of about 35 and 70 nT and a frequency of 7 Hz. These predictions were congruent with prior rat experiments using magnetic field strengths of about 1-5000 nT at 0.5, 7, 40, or 60 Hz. The model proposes that weaker fields in the nanoTesla to picoTesla range would optimally affect micromolar melatonin concentrations and that mitochondrial proton gradients may be critical. The model offers an alternative explanation to variations of Faraday's Law and the Boltzmann constant that have been used to dismiss biological effects from weak magnetic fields.
Exposure Information
Specific exposure levels were not quantified in this study.
Show BibTeX
@article{a_potential_multiple_resonance_mechanism_by_which_weak_magnetic_fields_affect_molecules_and_medical_problems_the_example_of_melatonin_and_experimental_multiple_sclerosis_ce1454,
author = {Unknown},
title = {A potential multiple resonance mechanism by which weak magnetic fields affect molecules and medical problems: the example of melatonin and experimental "multiple sclerosis"},
year = {2006},
doi = {10.1016/J.MEHY.2005.09.044},
}