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Note: This study found no significant biological effects under its experimental conditions. We include all studies for scientific completeness.

Effects of 60-Hz fields, estradiol and xenoestrogens on human breast cancer cells

No Effects Found

Authors not listed · 1996

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These studies suggest that estrogen and xenoestrogens stimulate growth-arrested breast cancer cells to enter the growth cycle, but EMF exposure does not.

Plain English Summary

Summary written for general audiences

Exposure of breast cancer cells to 12 mG, 1 G, or 9 G 60-Hz electromagnetic fields failed to stimulate Cdk2 or cyclin B1-cdc2 activity or cyclin D1 synthesis. Simultaneous co-exposure to 60-Hz fields and chemical promoters did not enhance Cdk2 activation beyond the effect of the chemical promoter alone. Estrogen and xenoestrogens stimulated estrogen receptor binding to the estrogen receptor element, but EMF did not.

Why This Matters

These studies suggest that estrogen and xenoestrogens stimulate growth-arrested breast cancer cells to enter the growth cycle, but EMF exposure does not. EMF exposure does not appear to act as a promoter or DNA-damaging agent for human breast cancer cells in vitro.

Exposure Information

A logarithmic frequency spectrum from 10 Hz to 100 GHz showing where this study's 60 Hz exposure sits relative to common EMF sources.Where This Frequency Sits on the EMF SpectrumELFVLFLF / MFHF / VHFUHFSHFmm10 Hz100 GHzThis study: 60 HzCell phones~1 GHzWiFi2.4 GHz5G mm28 GHzLogarithmic scale

Specific exposure levels were not quantified in this study.

Cite This Study
Unknown (1996). Effects of 60-Hz fields, estradiol and xenoestrogens on human breast cancer cells.
Show BibTeX
@article{effects_of_60_hz_fields_estradiol_and_xenoestrogens_on_human_breast_cancer_cells_ce1591,
  author = {Unknown},
  title = {Effects of 60-Hz fields, estradiol and xenoestrogens on human breast cancer cells},
  year = {1996},
  doi = {10.2307/3579306},
  
}

Quick Questions About This Study

The study tested 60-Hz fields at 12 mG, 1 G, and 9 G on breast cancer cells.
Exposure to 60-Hz EMF at 12 mG, 1 G, or 9 G failed to stimulate Cdk2 or cyclin B1-cdc2 activity or cyclin D1 synthesis.
Simultaneous co-exposure to 60-Hz fields and chemical promoters did not enhance Cdk2 activation above the level produced by the chemical promoter alone.
EMF exposure does not appear to act as a promoter or DNA-damaging agent for human breast cancer cells in vitro.