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Note: This study found no significant biological effects under its experimental conditions. We include all studies for scientific completeness.

Cardiovascular

Savchenko L, Martinelli I, Marsal D, Zhdan V, Tao J, Kunduzova O

No Effects Found

Authors not listed · 2023

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28 days of cell phone frequency EMF altered heart cell energy systems without causing obvious damage.

Plain English Summary

Summary written for general audiences

Researchers exposed mice to 915 MHz electromagnetic fields (similar to cell phone frequencies) for 28 days and examined heart muscle effects. They found changes in cellular energy production systems within heart mitochondria, but no structural heart damage or oxidative stress markers. The study suggests EMF exposure can alter how heart cells generate energy, though the health implications remain unclear.

Exposure Information

A logarithmic frequency spectrum from 10 Hz to 100 GHz showing where this study's 915 MHz exposure sits relative to common EMF sources.Where This Frequency Sits on the EMF SpectrumELFVLFLF / MFHF / VHFUHFSHFmm10 Hz100 GHzThis study: 915 MHzPower lines50/60 Hz5G mm28 GHzLogarithmic scale
Cite This Study
Unknown (2023). Savchenko L, Martinelli I, Marsal D, Zhdan V, Tao J, Kunduzova O.
Show BibTeX
@article{savchenko_l_martinelli_i_marsal_d_zhdan_v_tao_j_kunduzova_o_ce3009,
  author = {Unknown},
  title = {Savchenko L, Martinelli I, Marsal D, Zhdan V, Tao J, Kunduzova O},
  year = {2023},
  doi = {10.3389/fcvm.2023.1205893},
  
}
DOI: 10.3389/fcvm.2023.1205893PubMed: 37351281

Quick Questions About This Study

Yes, this study found that 28 days of 915 MHz electromagnetic field exposure significantly increased protein expression in cardiac mitochondrial complexes I-IV, indicating changes to cellular energy production systems in mouse hearts.
No structural abnormalities were identified in the left ventricles of mice exposed to 915 MHz EMF for 28 days. The hearts appeared functionally and structurally normal despite mitochondrial changes.
No, plasma malondialdehyde levels and cardiac superoxide dismutase 2 expression remained unchanged after 28 days of 915 MHz EMF exposure, indicating no increase in oxidative stress markers.
The study found that while other mitochondrial complex proteins increased with EMF exposure, the α-subunit of ATP synthase (Complex V) expression remained stable across all groups.
Yes, measurements using the Seahorse XF24 analyzer demonstrated that prolonged 915 MHz electromagnetic stress modified mitochondrial respiratory capacity in isolated cardiac mitochondria from exposed mice.