6,486 Studies Reviewed. 85.2% of Original Studies Found Biological Effects. The Evidence is Clear.
Shield Your Body
Reproductive Health

Radiofrequency electromagnetic radiation from cell phone causes defective testicular function in male Wistar rats.

Bioeffects Seen

Oyewopo AO, Olaniyi SK, Oyewopo CI, Jimoh AT. · 2017

View Original Abstract
Share:

Chronic exposure to cell phone radiofrequency radiation for 28 days caused defective testicular function with increased oxidative stress and decreased gonadotropic hormones in male Wistar rats.

Plain English Summary

Summary written for general audiences

Researchers randomly assigned adult male Wistar rats to a control group, a sham exposure group, and groups exposed to cell phone radiation for 1, 2, or 3 hours over a 28-day period. Histomorphometry showed no significant change in germinal epithelial diameter or cross-sectional diameter in any exposure group compared with controls. Group D showed a significantly smaller lumen diameter than Group B, and all exposure groups showed uneven germinal epithelial distribution with epithelial degeneration in Group D. Sera malondialdehyde increased and superoxide dismutase decreased in all exposure groups, while follicle-stimulating hormone, luteinizing hormone, and testosterone decreased significantly in groups C and D.

Why This Matters

The study aimed to investigate how cell phone radiation affects testicular structure and biochemistry. The researchers performed histomorphometric, biochemical, and histological examinations. They concluded that chronic radiofrequency exposure leads to defective testicular function associated with increased oxidative stress and decreased gonadotropic hormones.

Exposure Information

Specific exposure levels were not quantified in this study. Duration: 1-hr, 2-hr, 3-hr exposure

Study Details

The current study was undertaken to investigate the effects of the emitted radiation by cell phones on testicular histomorphometry and biochemical analyses

Adult male Wistar rats weighing 180-200 g were randomly allotted to control, group A (switched off m...

The histomorphometric parameters showed no significant change (p < .05) in the levels of germinal ep...

The study demonstrates that chronic exposure to radiofrequency electromagnetic radiation of cell phone leads to defective testicular function that is associated with increased oxidative stress and decreased gonadotropic hormonal profile.

Cite This Study
Oyewopo AO, Olaniyi SK, Oyewopo CI, Jimoh AT. (2017). Radiofrequency electromagnetic radiation from cell phone causes defective testicular function in male Wistar rats. Andrologia. 2017 Dec;49(10). doi: 10.1111/and.12772. Epub 2017 Mar 6.
Show BibTeX
@article{ao_2017_radiofrequency_electromagnetic_radiation_from_1689,
  author = {Oyewopo AO and Olaniyi SK and Oyewopo CI and Jimoh AT.},
  title = {Radiofrequency electromagnetic radiation from cell phone causes defective testicular function in male Wistar rats.},
  year = {2017},
  
  url = {https://pubmed.ncbi.nlm.nih.gov/28261838/},
}
PubMed: 28261838

Cited By (53 papers)

View all 53 citing papers on Semantic Scholar

Quick Questions About This Study

Adult male Wistar rats were exposed to cell phone radiofrequency radiation for 1, 2, or 3 hours over a 28-day period, with a sham group exposed to a switched-off phone.
Germinal epithelial diameter and cross-sectional diameter did not change significantly in any exposure group compared to controls. Group D showed a significantly smaller lumen diameter than Group B, and all exposure groups showed uneven germinal epithelial distribution with epithelial degeneration in Group D.
Malondialdehyde increased and superoxide dismutase decreased in all exposure groups compared to controls, indicating increased oxidative stress.
Follicle-stimulating hormone, luteinizing hormone, and testosterone decreased significantly in groups C and D compared to controls.
Chronic exposure to cell phone radiofrequency radiation led to defective testicular function associated with increased oxidative stress and decreased gonadotropic hormones.