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Exposure to electromagnetic field attenuates oxygen-glucose deprivation-induced microglial cell death by reducing intracellular Ca2+ and ROS.

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Duong CN, Kim JY · 2016

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Laboratory study shows 50 Hz magnetic fields at therapeutic levels can protect brain cells from oxygen deprivation damage.

Plain English Summary

Summary written for general audiences

Researchers exposed human brain immune cells to magnetic fields at 50 Hz while depriving them of oxygen to mimic stroke conditions. The magnetic field exposure protected cells from dying by reducing harmful calcium and oxidative stress, suggesting potential therapeutic applications for stroke treatment.

Why This Matters

This research presents an intriguing paradox in EMF science. While most studies focus on potential harmful effects of electromagnetic fields, this laboratory study suggests that specific frequencies and intensities might actually protect brain cells under certain conditions. The 1 mT magnetic field strength used here is significantly stronger than typical household exposures (which range from 0.01 to 0.2 mT near appliances), indicating this was a controlled therapeutic-level exposure rather than everyday environmental EMF. What makes this study particularly interesting is its precision - only the 50 Hz frequency at 1 mT showed strong protective effects, while other combinations provided little to no benefit. This specificity suggests that EMF bioeffects are highly dependent on exact parameters, not just presence or absence of fields. The findings add to growing evidence that EMF interactions with biological systems are far more nuanced than simple 'harmful' or 'harmless' categories suggest.

Exposure Details

Magnetic Field
1 mG
Source/Device
10, 50, or 100 Hz
Exposure Duration
4 hours

Exposure Context

This study used 1 mG for magnetic fields:

Building Biology guidelines are practitioner-based limits from real-world assessments. BioInitiative Report recommendations are based on peer-reviewed science. Check Your Exposure to compare your own measurements.

Where This Falls on the Concern Scale

Study Exposure Level in ContextA logarithmic scale showing exposure levels relative to Building Biology concern thresholds and regulatory limits.Study Exposure Level in ContextThis study: 1 mGExtreme Concern5 mGFCC Limit2,000 mGEffects observed in the Severe Concern range (Building Biology)FCC limit is 2,000x higher than this exposure level

Study Details

The aim of this research was to demonstrate the protective effects of electromagnetic field (EMF) exposure on the human microglial cell line, HMO6, against ischemic cell death induced by in vitro oxygen-glucose deprivation (OGD).

HMO6 cells were cultured for 4 h under OGD with or without exposure to EMF with different combinatio...

OGD caused significant HMO6 cell death as well as elevation of intracellular Ca2+ and ROS levels. Am...

50 Hz/1 mT EMF protects human microglial cells from OGD-induced cell death by interfering with OGD-induced elevation of intracellular Ca2+ and ROS levels, and xanthine oxidase is one of the main mediators involved in OGD-induced HMO6 cell death. Non-invasive treatment of EMF radiation may be clinically useful to attenuate hypoxic-ischemic brain injury.

Cite This Study
Duong CN, Kim JY (2016). Exposure to electromagnetic field attenuates oxygen-glucose deprivation-induced microglial cell death by reducing intracellular Ca2+ and ROS. Int J Radiat Biol. 92(4):195-201, 2016.
Show BibTeX
@article{cn_2016_exposure_to_electromagnetic_field_351,
  author = {Duong CN and Kim JY},
  title = {Exposure to electromagnetic field attenuates oxygen-glucose deprivation-induced microglial cell death by reducing intracellular Ca2+ and ROS.},
  year = {2016},
  doi = {10.3109/09553002.2016.1136851},
  url = {https://www.tandfonline.com/doi/abs/10.3109/09553002.2016.1136851},
}

Quick Questions About This Study

Researchers exposed human brain immune cells to magnetic fields at 50 Hz while depriving them of oxygen to mimic stroke conditions. The magnetic field exposure protected cells from dying by reducing harmful calcium and oxidative stress, suggesting potential therapeutic applications for stroke treatment.