Note: This study found no significant biological effects under its experimental conditions. We include all studies for scientific completeness.
Epilepsy but not mobile phone frequency (900 MHz) induces apoptosis and calcium entry in hippocampus of epileptic rat: involvement of TRPV1 channels.
Nazıroğlu M, Özkan FF, Hapil SR, Ghazizadeh V, Çiğ B. · 2015
View Original AbstractCell phone radiation didn't worsen brain cell damage in epileptic rats, suggesting EMF may not amplify existing neurological conditions.
Plain English Summary
Researchers exposed brain cells from epileptic rats to 900 MHz cell phone radiation for one hour to see if it would worsen brain damage. While epilepsy itself caused significant cellular damage including cell death and calcium influx, the cell phone radiation did not add any additional harmful effects. This suggests that 900 MHz EMF exposure may not exacerbate existing brain conditions like epilepsy.
Study Details
We investigated the effects of mobile phone (900 MHz) EMR exposure on Ca2+ influx, apoptosis, oxidative stress, and TRPV1 channel activations in the hippocampus of pentylenetetrazol (PTZ)-induced epileptic rats.
Freshly isolated hippocampal neurons of twenty-one rats were used in study within three groups namel...
The apoptosis, mitochondrial membrane depolarization, intracellular reactive oxygen species (ROS), a...
In conclusion, apoptosis, mitochondrial, ROS, and Ca2+ influx via TRPV1 channel were increased in the hippocampal neurons by epilepsy induction although the mobile phone did not change the values. The results indicated that TRPV1 channels in hippocampus may possibly be a novel target for effective target of epilepsy.
Show BibTeX
@article{m_2015_epilepsy_but_not_mobile_2886,
author = {Nazıroğlu M and Özkan FF and Hapil SR and Ghazizadeh V and Çiğ B.},
title = {Epilepsy but not mobile phone frequency (900 MHz) induces apoptosis and calcium entry in hippocampus of epileptic rat: involvement of TRPV1 channels.},
year = {2015},
doi = {10.1007/s00232-014-9744-y},
url = {https://link.springer.com/article/10.1007/s00232-014-9744-y},
}