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Absence of corneal endothelium injury in non-human primates treated with and without ophthalmologic drugs and exposed to 2.8 GHz pulsed microwaves.

No Effects Found

Lu ST, D'Andrea J, Chalfin S, Crane C, Marchello D, Garay R, Hatcher D, Ziriax J. · 2010

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Intense microwave exposure showed no corneal damage in primates at levels hundreds of times higher than typical wireless device exposure.

Plain English Summary

Summary written for general audiences

Researchers exposed four rhesus monkeys to intense 2.8 GHz microwave radiation for 36 hours total over three weeks and measured any damage to the corneal endothelium (the inner layer of cells in the eye's cornea). The study found no changes in corneal cell density or thickness, even at power levels more than ten times higher than previous studies that reported eye damage. This suggests that microwave exposure at these levels may not harm this specific part of the eye.

Exposure Information

A logarithmic frequency spectrum from 10 Hz to 100 GHz showing where this study's 2.80 GHz exposure sits relative to common EMF sources.Where This Frequency Sits on the EMF SpectrumELFVLFLF / MFHF / VHFUHFSHFmm10 Hz100 GHzThis study: 2.80 GHzPower lines50/60 Hz5G mm28 GHzLogarithmic scale

The study examined exposure from: 2.8 GHz Duration: 4 h/day, 3 days/week for 3 weeks (nine exposures and 36 h total)

Study Details

The aim of this study is to investigate Absence of corneal endothelium injury in non-human primates treated with and without ophthalmologic drugs and exposed to 2.8 GHz pulsed microwaves.

In an attempt to confirm these observations, four adult male Rhesus monkeys (Macaca mulatta) under p...

Pulsed microwave exposure did not cause alterations in corneal endothelial cell density and corneal ...

Cite This Study
Lu ST, D'Andrea J, Chalfin S, Crane C, Marchello D, Garay R, Hatcher D, Ziriax J. (2010). Absence of corneal endothelium injury in non-human primates treated with and without ophthalmologic drugs and exposed to 2.8 GHz pulsed microwaves. Bioelectromagnetics.31(4):324-333,2010.
Show BibTeX
@article{st_2010_absence_of_corneal_endothelium_3213,
  author = {Lu ST and D'Andrea J and Chalfin S and Crane C and Marchello D and Garay R and Hatcher D and Ziriax J.},
  title = {Absence of corneal endothelium injury in non-human primates treated with and without ophthalmologic drugs and exposed to 2.8 GHz pulsed microwaves.},
  year = {2010},
  
  url = {https://www.researchgate.net/publication/41175517_Absence_of_corneal_endothelium_injury_in_non-human_primates_treated_with_and_without_ophthalmologic_drugs_and_exposed_to_28_GHz_pulsed_microwaves},
}

Quick Questions About This Study

A 2010 study exposed rhesus monkeys to intense 2.8 GHz microwave radiation for 36 hours over three weeks and found no damage to corneal endothelium cells. The research showed no changes in corneal cell density or thickness, even at power levels more than ten times higher than previous studies reporting eye damage.
Research on rhesus monkeys found that pulsed 2.8 GHz microwave exposure did not affect corneal thickness. The study measured corneal endothelium after 36 hours of total exposure over three weeks, finding no alterations in cell density or tissue thickness even at very high power levels.
The 2010 primate study tested whether ophthalmologic drugs offered protection against 2.8 GHz microwave radiation, but found no eye damage occurred with or without the drugs. This suggests the microwave exposure itself didn't harm the corneal endothelium, making drug protection unnecessary at these exposure levels.
The 2010 study used 2.8 GHz microwave radiation at power levels more than ten times higher than previous research that reported eye damage. Despite this intense exposure over 36 hours across three weeks, researchers found no damage to the corneal endothelium in rhesus monkeys.
The 2010 rhesus monkey study could not confirm previous reports of corneal damage from pulsed microwave exposure. Using 2.8 GHz radiation at power levels more than ten times higher than earlier studies, researchers found no changes in corneal endothelial cell density or thickness.