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Indication of cocarcinogenic potential of chronic UMTS-modulated radiofrequency exposure in an ethylnitrosourea mouse model.

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Tillmann T, Ernst H, Streckert J, Zhou Y, Taugner F, Hansen V, Dasenbrock C. · 2010

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Mice pretreated with ethylnitrosourea and exposed to UMTS at 4.8 W/m(2) showed increased lung tumour rates, more lung carcinomas, higher carcinoma multiplicity, and doubled metastasising lung tumours.

Plain English Summary

Summary written for general audiences

Researchers exposed mice to UMTS fields at 0, 4.8, and 48 W/m(2) for up to 24 months starting from embryo-fetal exposure, with the 4.8 W/m(2) group also receiving prenatal ethylnitrosourea at 40 mg ENU/kg body weight. The high-level UMTS exposure (48 W/m(2)) showed tumour incidences comparable to sham and cage control groups. Mice given ethylnitrosourea and exposed to 4.8 W/m(2) UMTS had higher lung tumour rates and more lung carcinomas than ethylnitrosourea-only controls. Lung carcinoma multiplicity increased and the number of metastasising lung tumours doubled in the combined exposure group.

Why This Matters

The authors described the work as a pilot study. They reported a cocarcinogenic effect of lifelong UMTS exposure at 4.8 W/m(2) in female B6C3F1 descendants pretreated with ethylnitrosourea.

Exposure Details

Power Density
0, 0.48, 4.8 mW/cm²

Where This Falls on the Concern Scale

Study Exposure Level in ContextStudy Exposure Level in ContextExtreme Concern - 1,000 µW/m²FCC Limit - 10M µW/m²

Study Details

To evaluate putative effects on tumour susceptibility in mice exposed to a UMTS (universal mobile telecommunications system) test signal for up to 24 months, commencing with embryo-fetal exposure.

Animals were exposed to UMTS fields with intensities of 0, 4.8, and 48 W/m(2), the low-dose group (4...

The high-level UMTS exposure (48 W/m(2)), the sham exposure, and the cage control groups showed comp...

This pilot study indicates a cocarcinogenic effect of lifelong UMTS exposure (4.8 W/m(2)) in female B6C3F1 descendants subjected to pretreatment with ethylnitrosourea.

Cite This Study
Tillmann T, Ernst H, Streckert J, Zhou Y, Taugner F, Hansen V, Dasenbrock C. (2010). Indication of cocarcinogenic potential of chronic UMTS-modulated radiofrequency exposure in an ethylnitrosourea mouse model. nt J Radiat Biol. 86(7):529-541, 2010.
Show BibTeX
@article{t_2010_indication_of_cocarcinogenic_potential_1365,
  author = {Tillmann T and Ernst H and Streckert J and Zhou Y and Taugner F and Hansen V and Dasenbrock C.},
  title = {Indication of cocarcinogenic potential of chronic UMTS-modulated radiofrequency exposure in an ethylnitrosourea mouse model.},
  year = {2010},
  
  url = {https://pubmed.ncbi.nlm.nih.gov/20545575/},
}
PubMed: 20545575

Cited By (70 papers)

View all 70 citing papers on Semantic Scholar

Quick Questions About This Study

Researchers exposed mice to UMTS fields at 0, 4.8, and 48 W/m(2) for up to 24 months starting from embryo-fetal exposure, with the 4.8 W/m(2) group also receiving prenatal ethylnitrosourea at 40 mg ENU/kg body weight. The study used female B6C3F1 descendants.
Mice given ethylnitrosourea and exposed to 4.8 W/m(2) UMTS had higher lung tumour rates, more lung carcinomas, increased carcinoma multiplicity, and the number of metastasising lung tumours doubled compared to ethylnitrosourea-only controls.
The high-level UMTS exposure (48 W/m(2)) showed tumour incidences comparable to sham and cage control groups.
The authors concluded that lifelong UMTS exposure at 4.8 W/m(2) has a cocarcinogenic effect in female B6C3F1 descendants pretreated with ethylnitrosourea.
The authors described it as a pilot study indicating a cocarcinogenic effect.