8,700 Studies Reviewed. 87.0% Found Biological Effects. The Evidence is Clear.

Chronic Exposure to a GSM-like Signal (Mobile Phone) Does Not Stimulate the Development of DMBA-Induced Mammary Tumors in Rats: Results of Three Consecutive Studies.

Bioeffects Seen

Bartsch H, Bartsch C, Seebald E, Deerberg F, Dietz K, Vollrath L, Mecke D. · 2002

View Original Abstract
Share:

Cell phone-level radiation did not promote breast tumors in rats, with one study actually showing delayed tumor development.

Plain English Summary

Summary written for general audiences

Scientists tested whether cell phone radiation affects breast cancer development in rats across three studies. The radiation did not increase tumor rates or speed cancer growth overall. One study showed slightly delayed tumor development, but this wasn't repeated. Results suggest no clear cancer risk.

Why This Matters

This study represents one of the more rigorous approaches to testing whether cell phone radiation promotes cancer development. The researchers used exposure levels (17.5-70 mW/kg SAR) that mirror what you experience during typical cell phone use, and they replicated their experiment three times to ensure reliability. What makes this research particularly valuable is that it tested whether RF radiation acts as a tumor promoter rather than a tumor initiator, addressing concerns that cell phone use might accelerate existing cancer processes. The finding that one study actually showed delayed tumor development in exposed animals is intriguing and suggests the biological effects may be more complex than simple promotion or inhibition. While these results are reassuring for breast cancer specifically, the authors correctly note that blood cancers like leukemia may respond differently to RF exposure and require separate investigation.

Exposure Details

SAR
0.0175 – 0.07 W/kg
Power Density
0.1 µW/m²
Source/Device
900 MHz pulsed at 217 Hz

Exposure Context

This study used 0.1 µW/m² for radio frequency:

Building Biology guidelines are practitioner-based limits from real-world assessments. BioInitiative Report recommendations are based on peer-reviewed science. Check Your Exposure to compare your own measurements.

Where This Falls on the Concern Scale

Study Exposure Level in ContextStudy Exposure Level in ContextThis study: 0.1 µW/m²Extreme Concern - 1,000 uW/m2FCC Limit - 10M uW/m2Effects observed in the Slight Concern rangeFCC limit is 100,000,000x higher than this level
A logarithmic frequency spectrum from 10 Hz to 100 GHz showing where this study's 217 Hz - 900 MHz exposure sits relative to common EMF sources.Where This Frequency Sits on the EMF SpectrumELFVLFLF / MFHF / VHFUHFSHFmm10 Hz100 GHzThis study: 217 Hz - 900 MHzPower lines50/60 HzCell phones~1 GHzWiFi2.4 GHz5G mm28 GHzLogarithmic scale

Study Details

In this study, an RF field used in mobile telecommunication was tested using 7,12-dimethylbenz[a]anthracene (DMBA)-induced mammary tumors in female Sprague-Dawley rats as a model for human breast cancer.

Three experiments were carried out under strictly standardized conditions and were started on the sa...

The overall results of the three studies are that there was no statistically significant effect of R...

These results show that low-level RF radiation does not appear to possess carcinogenic or cancer-promoting effects on DMBA-induced mammary tumors. To explain the mechanisms underlying the different results obtained in the three experiments, a hypothesis is presented which is based upon the neuroendocrine control mechanisms involved in the promotion of DMBA-induced mammary tumors. Despite the apparent absence of stimulatory effects of low-level RF-field exposure on the development and growth of solid tumors, it will be necessary to verify these results for leukemias and lymphomas, which may have completely different biological control mechanisms.

Cite This Study
Bartsch H, Bartsch C, Seebald E, Deerberg F, Dietz K, Vollrath L, Mecke D. (2002). Chronic Exposure to a GSM-like Signal (Mobile Phone) Does Not Stimulate the Development of DMBA-Induced Mammary Tumors in Rats: Results of Three Consecutive Studies. Radiat Res 157(2):183-190, 2002.
Show BibTeX
@article{h_2002_chronic_exposure_to_a_846,
  author = {Bartsch H and Bartsch C and Seebald E and Deerberg F and Dietz K and Vollrath L and Mecke D.},
  title = {Chronic Exposure to a GSM-like Signal (Mobile Phone) Does Not Stimulate the Development of DMBA-Induced Mammary Tumors in Rats: Results of Three Consecutive Studies.},
  year = {2002},
  
  url = {https://meridian.allenpress.com/radiation-research/article-abstract/157/2/183/331562/Chronic-Exposure-to-a-GSM-like-Signal-Mobile-Phone},
}

Quick Questions About This Study

No, 900 MHz cell phone radiation did not increase breast cancer rates in rats across three consecutive studies. The radiation showed no statistically significant effect on tumor development, with risk ratios of 1.08 for benign tumors and 0.96 for malignant tumors, indicating no clear cancer risk.
One study showed GSM signals delayed mammary tumor development in rats (278 days vs 145 days), but two follow-up experiments found no such effect. The delayed growth was not reproducible, suggesting no consistent protective effect from 900 MHz radiation exposure.
Scientists exposed rats to DMBA-induced mammary tumors while simultaneously exposing them to GSM-like 900 MHz radiation pulsed at 217 Hz. The study tracked tumor development over extended periods, with median tumor latency ranging from 145-278 days depending on the experiment.
Low-level RF radiation does not appear to have carcinogenic or cancer-promoting effects on DMBA-induced mammary tumors in rats. Three consecutive studies found no statistically significant impact on tumor incidence or growth rates from chronic GSM-like signal exposure.
Researchers hypothesize that neuroendocrine control mechanisms may explain why one experiment showed delayed tumor development while two others didn't. These mechanisms are involved in promoting DMBA-induced mammary tumors and could account for the inconsistent results across the three studies.