8,700 Studies Reviewed. 87.0% Found Biological Effects. The Evidence is Clear.
Shield Your Body

Note: This study found no significant biological effects under its experimental conditions. We include all studies for scientific completeness.

Cellular Effects

Mobile phone base station-emitted radiation does not induce phosphorylation of Hsp27.

No Effects Found

Hirose H, Sakuma N, Kaji N, Nakayama K, Inoue K, Sekijima M, Nojima T, Miyakoshi J. · 2007

View Original Abstract
Share:

Cell tower radiation at public safety limits did not trigger cellular stress responses in human cells during laboratory testing.

Plain English Summary

Summary written for general audiences

Japanese researchers exposed human brain and lung cells to radiofrequency radiation at levels similar to cell tower emissions (2.1 GHz) for up to 48 hours. They found no changes in heat shock proteins (cellular stress markers that increase when cells are damaged) even at exposure levels 10 times higher than public safety limits. This suggests that cell tower-level RF radiation does not trigger detectable cellular stress responses in laboratory conditions.

Exposure Information

A logarithmic frequency spectrum from 10 Hz to 100 GHz showing where this study's 2.14 GHz exposure sits relative to common EMF sources.Where This Frequency Sits on the EMF SpectrumELFVLFLF / MFHF / VHFUHFSHFmm10 Hz100 GHzThis study: 2.14 GHzPower lines50/60 Hz5G mm28 GHzLogarithmic scale

The study examined exposure from: 2.1425 GHz Duration: 2-48 hours

Study Details

An in vitro study focusing on the effects of low-level radiofrequency (RF) fields from mobile radio base stations employing the International Mobile Telecommunication 2000 (IMT-2000) cellular system was conducted to test the hypothesis that modulated RF fields act to induce phosphorylation and overexpression of heat shock protein hsp27

First, we evaluated the responses of human cells to microwave exposure at a specific absorption rate...

Under the RF field exposure conditions described above, no significant differences in the expression...

Our results confirm that exposure to low-level RF field up to 800 mW/kg does not induce phosphorylation of hsp27 or expression of hsp gene family.

Cite This Study
Hirose H, Sakuma N, Kaji N, Nakayama K, Inoue K, Sekijima M, Nojima T, Miyakoshi J. (2007). Mobile phone base station-emitted radiation does not induce phosphorylation of Hsp27. Bioelectromagnetics.28(2):99-108, 2007.
Show BibTeX
@article{h_2007_mobile_phone_base_stationemitted_3086,
  author = {Hirose H and Sakuma N and Kaji N and Nakayama K and Inoue K and Sekijima M and Nojima T and Miyakoshi J.},
  title = {Mobile phone base station-emitted radiation does not induce phosphorylation of Hsp27.},
  year = {2007},
  
  url = {https://pubmed.ncbi.nlm.nih.gov/17004241/},
}
PubMed: 17004241

Cited By (40 papers)

Quick Questions About This Study

A 2007 Japanese study found that 2.1 GHz radiation from cell towers does not cause cellular stress. Researchers exposed human brain and lung cells for up to 48 hours at levels 10 times higher than public safety limits and detected no stress protein changes.
No, cell tower frequencies do not trigger heat shock proteins according to laboratory research. Scientists exposed human cells to 2.1 GHz radiation similar to cell tower emissions and found no activation of these cellular stress markers even at high exposure levels.
Research shows 800 mW/kg RF exposure does not harm human cells. A controlled study exposed brain and lung cells to this level of 2.1 GHz radiation for 48 hours without detecting cellular damage or stress responses.
W-CDMA cell tower signals do not damage brain cells based on laboratory evidence. Japanese researchers found no cellular stress responses in human brain cells exposed to these 3G network frequencies, even at exposure levels exceeding public safety limits.
Cells can be safely exposed to 2.1 GHz radiation for at least 48 hours according to research findings. Scientists detected no cellular damage or stress protein activation in human brain and lung cells during this extended exposure period.