Unknown authors · 2010
A case-control study in southwestern France from May 1999 to April 2001 included 221 adults with central nervous system tumors and 442 matched controls. Occupational ELF and radiofrequency exposure was assessed by expert judgment using job calendars, and residential exposure by distance to power lines via a geographic information system. Occupational ELF exposure showed a significant increase in meningioma risk (OR=3.02, 95% CI 1.10-8.25), while living within 100 m of power lines gave a non-significant increase (OR=2.99, 95% CI 0.86-10.40).
Unknown authors · 2010
Researchers conducted a measurement campaign aboard Italian main line trains to assess railway workers' exposure to static and ELF magnetic fields. Measurements were taken during regular service with two engine drivers present, in all accessible areas, across routes from tens to hundreds of kilometres. The protocol used broadband metres and personal metres for individual exposure assessment across seven different engine or train models. Traction motors were fed by AC current except for two engines where AC powered only auxiliary services. Average static field exposure was slightly above background geomagnetic levels, occasionally reaching millitesla, while average ELF exposure was 1-2 μT with hot spots near wiring reaching tens of microtesla.
Unknown authors · 2010
Previous epidemiological studies of radiofrequency electromagnetic fields relied on crude exposure proxies like job titles and equipment use. The Royal Norwegian Navy measured RF fields from antennas and radars at crew locations on fast patrol boats. The boats are small, placing crew close to RF-emitting equipment. Researchers described the measured exposure and proposed calculation methods using linear and spatial averages, percentage of ICNIRP, and squared deviation of ICNIRP.
Unknown authors · 2010
Researchers analyzed brain tumor data from 2000-2004 and 2007 cell phone subscription data. They found a strong correlation between cell phone subscriptions and brain tumor incidence (r = 0.950, P < 0.001). Multiple linear regression showed the association remained significant after controlling for population, mean family income, and mean age (P = 0.017). The authors considered the linear relationship disturbing and called for further epidemiological evaluation. They suggested it would be prudent to limit exposure to all sources of electromagnetic radiation.
Unknown authors · 2010
Long-term mobile phone users had higher incidence of brain tumors, parotid gland tumors, and seminoma, with odds ratios from 1.3 to 6.1 for ipsilateral use. Current safety limits are based only on thermal mechanisms, but experimental data show metabolic changes from non-thermal exposure. Reproducible non-thermal effects include reactive oxygen species overproduction, heat shock protein expression, DNA damage, and apoptosis.
Unknown authors · 2010
Researchers are recruiting 250,000 men and women aged 18 years or older in Denmark, Finland, Sweden, the Netherlands, and UK. Participants will be followed up for 25+ years. Mobile telephone use is collected prospectively through questionnaires and objective traffic data from network operators. Associations with disease risks will be studied by linking cohort members to existing disease registries. Changes in symptoms such as headache and sleep quality and of general well-being are assessed by baseline and follow-up questionnaires.
Unknown authors · 2010
Divan H et al et al. · 2010
Researchers analysed a new dataset of DNBC children to see if a larger, separate group would produce similar results after considering additional confounders. The highest odds ratio for behavioural problems was found in children with both prenatal and postnatal cell phone exposure compared to those unexposed, with an adjusted effect estimate of 1.5 (95% CI 1.4 to 1.7). The findings replicated the previous publication, showing that cell phone use was associated with behavioural problems at age 7 years, and this association was not limited to early users of the technology. Although weaker in the new dataset, the associations remained even with further control for an extended set of potential confounders.
Unknown authors · 2010
The study is a re-analysis of risk for glioma in relation to mobile telephone use. The study compares its results with the Interphone international case-control study.
Yildirim MS, Yildirim A, Zamani AG, Okudan N. · 2010
The study evaluated the effects of mobile phone base stations on micronucleus frequency and chromosomal aberrations in blood cells of people living near base stations and healthy controls. Micronucleus frequency was 8.96 +/- 3.51 in the study group and 6.97 +/- 1.52 in controls (p: 0.16). Chromosomal aberrations were 0.36 +/- 0.31 in the study group and 0.75 +/- 0.61 in controls (p: 0.07). There was no significant difference in either measure between the two groups. The authors concluded that cellular phones and their base stations do not produce important carcinogenic changes.
Sekijima M et al. · 2010
The study examined cell proliferation and gene expression in three human cell lines after exposure to 2.1425 GHz continuous wave and W-CDMA modulated radiofrequency fields. Cells were exposed at specific absorption rates of 80, 250, or 800 mW/kg for up to 96 hours. No significant differences in cell growth or viability were found between exposed groups and sham controls, and less than 1% of genes showed altered expression.
Röösli M, Frei P, Mohler E, Hug K · 2010
The systematic review included 5 randomized human laboratory trials and 12 epidemiological studies. Most of the papers examined self-reported non-specific symptoms of ill-health. Most randomized trials did not detect an association between MPBS radiation and acute symptom development, and sporadic associations lacked a consistent pattern. The authors concluded that evidence for no acute effect up to 10 volts per metre is strong, but data are insufficient for long-term low-level exposure.
Redmayne M, Inyang I, Dimitriadis C, Benke G, Abramson MJ · 2010
The MoRPhEUS study included cordless phone access and use among 317 Year 7 students recruited from Melbourne, Australia. Researchers found a statistically significant positive relationship between cordless and mobile phone use (r = 0.38, p < 0.01). 87% of participants had a cordless phone at home and 77% owned a mobile phone.
Parazzini M et al. · 2010
Researchers investigated the potential effects of UMTS exposure at a high specific absorption rate on the human auditory system in 73 healthy young adults. Participants received a 20-minute UMTS exposure at 1947 MHz with a maximum SAR of 1.75 W/kg in a double-blind comparison of genuine and sham exposure. No consistent pattern of effects was found on hearing thresholds, otoacoustic emissions, or auditory evoked potentials. The study concluded that short-term UMTS exposure at this SAR does not cause measurable immediate effects on the human auditory system.
Lu ST et al. · 2010
Researchers exposed four adult male Rhesus monkeys to a 2.8 GHz pulsed microwave signal at 13.0 mW/cm(2) spatial and temporal average power density. The exposure raised eyelid temperature by 1.0-1.2 degrees C. Experiment I consisted of nine 4-hour exposures over three weeks.
Kowalczuk C et al. · 2010
Researchers used a doubly resonant cavity to test whether biological samples generate a second harmonic when exposed to continuous wave RF fields at 883 MHz. They tested over 500 samples including human and mouse cell suspensions, adherent cell lines, and mouse tissue slices, both viable and non-viable. Input powers were 0.1 or 1 mW, and the system could detect second harmonic signals above a noise floor of -169 dBm. No consistent second harmonic was detected, so the results do not support the hypothesis that living cells can demodulate RF energy.
Kim KB et al. · 2010
Researchers exposed MCF7 human breast cancer cells to 849 MHz RF radiation for one hour per day over three consecutive days at SARs of 2 or 10 W/kg. Protein expression profiles were analyzed using two-dimensional gel electrophoresis and showed no significant or reproducible changes after exposure. Follow-up analysis of three non-reproducible protein spots using mass spectrometry, RT-PCR, and Western blot confirmed no alterations in mRNA or protein levels.
The INTERPHONE Study Group. · 2010
An interview-based case-control study with 2708 glioma and 2409 meningioma cases and matched controls was conducted in 13 countries using a common protocol. Ever regular mobile phone use was associated with reduced odds ratios for glioma (OR 0.81, 95% CI 0.70-0.94) and meningioma (OR 0.79, 95% CI 0.68-0.91), possibly reflecting participation bias. No elevated odds ratio was observed 10 or more years after first phone use for either tumor type. In the highest decile of recalled cumulative call time (≥1640 hours), the odds ratio for glioma was 1.40 (95% CI 1.03-1.89) and for meningioma was 1.15 (95% CI 0.81-1.62), but the authors noted implausible values of reported use in this group.
Inskip PD, Hoover RN, Devesa SS. · 2010
Researchers examined temporal trends in brain cancer incidence rates in the United States using SEER program data from 1992 to 2006. Trends were downward or flat for all age groups except 20- to 29-year-olds, where females showed a statistically significant increase. The increase in young women was driven by frontal lobe cancers, while cancers in brain regions more exposed to phone radiation did not increase. The authors concluded that the incidence data do not support the view that cellular phone use causes brain cancer.
Hintzsche H, Stopper H. · 2010
Researchers collected buccal mucosa cells from 131 individuals and grouped them by mobile phone use. Cells were stained with alpha-tubulin-antibody and chromomycin A3, and micronuclei were counted in 2000 cells per person. Mobile phone use did not significantly increase micronucleus frequency.
Falzone N, Huyser C, Franken DR, Leszczynski D. · 2010
Researchers examined effects of mobile phone radiation on apoptosis-related properties in human spermatozoa. Ejaculated, density-purified, highly motile human spermatozoa were exposed to mobile phone radiation at specific absorption rates of 2.0 and 5.7 W/kg. Mobile phone radiation had no statistically significant effect on any of the parameters studied, including caspase 3 activity, externalization of phosphatidylserine, DNA strand breaks, and reactive oxygen species generation. This suggests that impairment of fertility reported in some studies was not caused by induction of apoptosis in spermatozoa.
Elliott P et al. · 2010
The study was a case-control study using cancer registry and national birth register data in Great Britain with 1397 cases aged 0-4 and 5588 controls matched by sex and date of birth. Mean distance from a macrocell base station, total power output within 700 m, and modelled power density at the birth address were similar for cases and controls. Adjusted odds ratios for all cancers were 1.01 (0.87-1.18) for intermediate and 1.02 (0.88-1.20) for highest modelled power density exposure, with no significant trends for brain and central nervous system cancers or leukaemia and non-Hodgkin's lymphoma.
Bourthoumieu S et al. · 2010
Researchers exposed human amniotic cells to GSM-900 MHz radiofrequency radiation at an average SAR of 0.25 W/kg for 24 hours. Genotoxic effects were assessed immediately and 24 hours after exposure using R-banded karyotyping of 100 metaphase cells per assay across four samples. No direct cytogenetic effects were observed at either time point.
Luukkonen J, Juutilainen J, Naarala J. · 2010
Researchers aimed to investigate cooperative effects of 872 MHz RF radiation and ferrous chloride on ROS production and DNA damage in human neuroblastoma SH-SY5Y cells. They exposed the cells to 872 MHz RF radiation at 5 W/kg SAR using continuous wave or GSM-like modulated signals for 1 hour in ROS experiments and 3 hours in comet assay experiments. Chemical treatments with ferrous chloride produced statistically significant responses, but RF radiation alone or combined with chemicals did not affect ROS production, DNA damage, or cell viability.
Okano T et al. · 2010
Researchers used a double-blind, counterbalanced crossover design with 10 normal subjects. Subjects performed antisaccade, cued saccade, and two overlap saccade tasks before and after 30 minutes of real or sham mobile phone exposure.
